Article Figures & Data
Figures
- Fig 1.
Case 1. Infarctlike lesion in a 74-year-old man. A, Axial T2WI shows hyperintense spots in the bilateral watershed area (white arrowheads). B, The corresponding areas show diffusion restriction on DWI (white arrows). C, Pathologic specimen shows irregular-shaped necrosis (double black arrow) and congestion (thick black arrows) surrounding the occluded vessel (arrowhead) (hematoxylin-eosin, original magnification ×25).
- Fig 2.
Case 2. Nonspecific white matter lesion in a 69-year-old man. A, Axial T2WI shows diffuse hyperintensity in the bilateral periventricular areas before treatment (white arrows). B, Posttreatment T2WI on day 94 shows a decrease in the abnormal signal intensity. C, Pathologic specimen shows occlusion of the vessels by tumor cells (black arrows) and diffuse demyelination (D) with or without evidence of infarction (Klüver-Barrera, original magnification ×25).
- Fig 3.
Case 2. Meningeal enhancement in a 69-year-old man. A, Gadolinium-enhanced coronal T1-weighted image shows abnormal meningeal enhancement around the temporal lobe before treatment (white arrowheads). B, Pathologic specimen shows thickening of the affected vessel walls with intraluminal (white arrow) and subendothelial (black arrowheads) tumoral infiltration (hematoxylin-eosin, original magnification ×25).
- Fig 4.
Case 4. Masslike lesion in a 70-year-old man. A, Coronal gadolinium-enhanced T1-weighted image shows ringlike enhancement before treatment (white arrow). B, Axial DWI shows no abnormal signal intensity in the lesion. C, Follow-up gadolinium-enhanced coronal T1-weighted image shows regression of the enhancement after treatment on day 121.
- Fig 5.
Case 8. Hyperintense lesion in the pons on a T2-weighted image in a 63-year-old woman. A, Axial T2WI shows symmetric hyperintense lesions in the center of the pons sparing the pontine tegmentum and ventrolateral pons (white arrow). DWI showed T2 shinethrough without diffusion restriction (not shown). B, T2WI after chemotherapy shows a decreased abnormal signal intensity in the pons on day 85 (white arrowhead).
- Fig 6.
Case 3. Tumor cell infiltration into the pontine vessels in an 84-year-old man. Pathologic specimen shows lymphoma cell infiltration into the capillaries in the pons (black arrows) (hematoxylin-eosin, original magnification ×50).
Tables
Case Tumoral Vascular Occlusion Infarction Demyelination Meningitis Tumor Cell Extravasation 1 X X/multiple hemorrhagic infarcts with necrosis and surrounding congestion X/cerebral WM 2 X (including pons) WM X/multiple hemorrhagic infarcts, thickening and subendothelial tumoral infiltration X/cerebral/cerebellar X/vascular wall 3 X (including pons) X/multiple hemorrhagic infarcts with necrosis and surrounding congestion X/cerebral WM X 5 X X/multiple infarcts X/cerebral WM (Not applicable to the corresponding area) 10 No tumor cell infiltration detected -
Note:—X indicates presence of finding.
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