PT  - JOURNAL ARTICLE
AU  - Cuvinciuc, V.
AU  - Viguier, A.
AU  - Calviere, L.
AU  - Raposo, N.
AU  - Larrue, V.
AU  - Cognard, C.
AU  - Bonneville, F.
TI  - Isolated Acute Nontraumatic Cortical Subarachnoid Hemorrhage
AID  - 10.3174/ajnr.A1986
DP  - 2010 Sep 01
TA  - American Journal of Neuroradiology
PG  - 1355--1362
VI  - 31
IP  - 8
4099  - http://www.ajnr.org/content/31/8/1355.short
4100  - http://www.ajnr.org/content/31/8/1355.full
SO  - Am. J. Neuroradiol.2010 Sep 01; 31
AB  - SUMMARY: Our aim was to review the etiologic background of isolated acute nontraumatic cSAH. While SAH located in the basal cisterns originates from a ruptured aneurysm in approximately 85% of cases, a broad spectrum of vascular and even nonvascular pathologies can cause acute nontraumatic SAH along the convexity. Arteriovenous malformations or fistulas, cortical venous and/or dural sinus thrombosis, and distal and proximal arteriopathies (RCVS, vasculitides, mycotic aneurysms, Moyamoya, or severe atherosclerotic carotid disease) should be sought by noninvasive imaging methods or/and conventional angiography. Additionally, PRES may also be a source of acute cSAH. In elderly patients, cSAH might be attributed to CAA if numerous hemorrhages are demonstrated by GRE T2 images. Finally, cSAH is rarely observed in nonvascular disorders, such as abscess and primitive or secondary brain tumors. CAAcerebral amyloid angiopathycSAHcortical subarachnoid hemorrhageCTACT angiographyCTVCT venographyCVTcerebral venous thrombosisDSAdigital subtraction angiographyDWIdiffusion-weighted imagingFLAIRfluid-attenuated inversion recoveryGdgadoliniumGRE T2gradient echo T2-weighted imagingMRAMR angiographyMRVMR venographyPRESposterior reversible encephalopathy syndromeRCVSreversible cerebral vasoconstriction syndromeSAHsubarachnoid hemorrhageSWIsusceptibility-weighted imagingTIAtransient ischemic attackTOFtime of flight